Module 2- PCOS, IGT, Insulin Resistance
Metabolic dysfunction, insulin resistance, and impaired glucose regulation are frequently overlooked contributors to low milk supply.
In this module, we’ll explore how PCOS, insulin resistance, impaired glucose tolerance (IGT), and metabolic patterns may influence mammary gland function, hormonal signaling, milk production regulation, and lactation outcomes.
We’ll also discuss common clinical presentations, contributing physiological mechanisms, assessment considerations, and root-cause patterns that may warrant further investigation in complex low milk supply cases.
Please move through the lesson slides first, follow along with the downloadable companion guide, and complete the knowledge check at the end of the module before progressing further through the system.
Estimated Completion Time: 45–60 minutes
Companion Guide
Download the companion guide for this lesson to follow along, review key concepts, and organize important clinical considerations discussed throughout the module.
Module Reflection
Before moving forward to the knowledge check, take a moment to reflect on the patterns and concepts discussed throughout this module.
Consider:
• How insulin resistance may impact lactation physiology and milk production regulation
• The relationship between PCOS, hormonal signaling, and mammary gland development
• Clinical patterns that may suggest underlying metabolic dysfunction
• Why some low milk supply cases may not respond to generic lactation recommendations alone
• The importance of considering root-cause physiology in complex lactation presentations
Module 2 Knowledge Check
Please complete the following knowledge check before continuing to the next module.
This assessment is designed to reinforce understanding of the key concepts discussed throughout the lesson.